Human ZMYND10/BLU/CILD22 ORF/cDNA clone-Lentivirus plasmid (NM_015896.2)

SKU: pGMLV000114
Size: 10 µg
Concentration: generally 0.5ug/ul, usually not less than 0.3ug/ul
Leading Time: 3-7 working days

Pre-made Human ZMYND10/BLU/CILD22 Lentiviral expression plasmid for ZMYND10 lentivirus packaging, ZMYND10 lentivirus production, overexpression stable cell line development, cell transient transfection and gene delivery targeting T/B/NK cells, macrophages, cardiomyocytes, hepatocytes, and neurons.

Our GM-Lentiviral vector is seamlessly integrated with the GM lentivirus packaging system. Discover more about the GM lentivirus packaging system.

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Product Description

Catalog ID pGMLV000114
Gene Name ZMYND10
Accession Number NM_015896.2
Gene ID 51364
Species Human
Product Type Lentivirus plasmid (overexpression)
Insert Length 1323 bp
Gene Alias BLU,CILD22,FLU
Fluorescent Reporter ZsGreen
Mammalian Cell Selection Puromyocin
Fusion Tag 3xflag (C-Terminal)
Promoter CMV
Resistance Amplicin

Reference




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    Associated products


    Category Cat No. Products Name
    Target Antibody GM-Tg-g-IP2820-Ab Anti-ZMYND10 monoclonal antibody
    Target Antigen GM-Tg-g-IP2820-Ag ZMYND10 protein
    ORF Viral Vector pGMLV000114 Human ZMYND10 Lentivirus plasmid
    ORF Viral Vector vGMLV000114 Human ZMYND10 Lentivirus particle


    Target information

    Target ID GM-IP2820
    Target Name ZMYND10
    Gene ID 51364, 114602, 702678, 363139, 101094912, 476615, 528799, 100146221
    Gene Symbol and Synonyms BLU,CILD22,DNAAF7,FLU,ZMYND10
    Uniprot Accession O75800
    Uniprot Entry Name ZMY10_HUMAN
    Protein Sub-location Introcelluar Protein
    Category Not Available
    Disease Cancer
    Gene Ensembl ENSG00000004838
    Target Classification Tumor-associated antigen (TAA)

    This gene encodes a protein containing a MYND-type zinc finger domain that likely functions in assembly of the dynein motor. Mutations in this gene can cause primary ciliary dyskinesia. This gene is also considered a tumor suppressor gene and is often mutated, deleted, or hypermethylated and silenced in cancer cells. Alternative splicing results in multiple transcript variants. [provided by RefSeq, Aug 2015]



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